A second site Kras(G12D) mutation that impairs PI3K binding rescues embryonic lethality, abrogates myeloproliferative disease, and delays lung tumorigenesis.



Wong, Jasmine C, Perez-Mancera, Pedro A, Kim, Jangkyung, Jen, Kuang-Yu, Kogan, Scott C, Firestone, Ari J, Collisson, Eric A, Tuveson, David A and Shannon, Kevin
(2020) A second site Kras(G12D) mutation that impairs PI3K binding rescues embryonic lethality, abrogates myeloproliferative disease, and delays lung tumorigenesis. .

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A second site Kras(G12D) mutation that impairs PI3K binding rescues embryonic lethality, abrogates myeloproliferative disease, and delays lung tumorigenesis.AACR Targeting RAS 2018 abstract.pdf - Accepted Version

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Item Type: Conference or Workshop Item (Unspecified)
Depositing User: Symplectic Admin
Date Deposited: 11 Sep 2020 07:37
Last Modified: 03 May 2021 00:10
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URI: https://livrepository.liverpool.ac.uk/id/eprint/3100707