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Hapangama, DK ORCID: 0000-0003-0270-0150, Drury, J ORCID: 0000-0002-2275-6664, Da Silva, L, Al-Lamee, H, Earp, A, Valentijn, AJ, Edirisinghe, DP, Murray, PA, Fazleabas, AT and Gargett, CE
(2019)
Abnormally located SSEA1+/SOX9+ endometrial epithelial cells with a basalis-like phenotype in the eutopic functionalis layer may play a role in the pathogenesis of endometriosis.
Human Reproduction, 34 (1).
pp. 56-68.
Button, L, Alnafakh, R, Drury, J ORCID: 0000-0002-2275-6664, DeCruze, SB, Saretzki, G, Adishesh, M and Hapangama, D ORCID: 0000-0003-0270-0150
(2019)
EXAMINATION OF GENES ENCODING TELOMERASE ASSOCIATED PROTEINS SUGGESTS A PROGNOSTIC RELEVANCE FOR NHP2 AND NOP10 IN ENDOMETRIAL CANCER.
In: ESGO Annual Meeting Abstracts.
Maclean, A, Drury, J ORCID: 0000-0002-2275-6664 and Hapangama, D ORCID: 0000-0003-0270-0150
(2017)
Identification of markers of endometrial epithelial cells (EEC's) contributes to the characterisation of EEC subtypes.
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Al-Nafakh, R, Adishesh, M, Drury, J ORCID: 0000-0002-2275-6664, Saretzki, G and Hapangama, D ORCID: 0000-0003-0270-0150
(2018)
Telomerase component, Dyskerin (DKC1), is expressed and hormonally regulated in healthy endometrium: implications for endometrial pathologies.
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Button, L, Adishesh, M, Drury, J ORCID: 0000-0002-2275-6664, DeCruze, SB and Hapangama, D ORCID: 0000-0003-0270-0150
(2019)
WARM ISCHAEMIA IS AN IMPORTANT PRE-ANALYTICAL VARIABLE AFFECTING THE ANALYSIS OF ENDOMETRIAL BIOSPECIMENS.
In: ESGO Annual Meeting Abstracts.
Maclean, A, Adishesh, M, Button, L, Richards, L, Alnafakh, R, Newton, E, Drury, J ORCID: 0000-0002-2275-6664 and Hapangama, DK ORCID: 0000-0003-0270-0150
(2022)
The effect of pre-analytical variables on downstream application and data analysis of human endometrial biopsies.
HUMAN REPRODUCTION OPEN, 2022 (3).
hoac026-.
Maclean, A, Bunni, E, Makrydima, S, Withington, A, Drury, J ORCID: 0000-0002-2275-6664 and Hapangama, D ORCID: 0000-0003-0270-0150
(2018)
The human fallopian tube vs endometrium: can differences in function and steroid hormone receptor expression be explained by differential hormone responsiveness?
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